Etiology
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Genetic (incidence by race, familial concordance, male:female = 2.5:1, FH in 1/4)
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Histologic (muscle ultrastructure, increased type I:II fiber ratio, cartilage defects, increased collagen synthesis, plantar flexion and varus deformity of talus, calcaneus, and cuboid.)
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theory of retraction fibrosis
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myofibroblast theory
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Vascular anomalies – hypoplasia or absence of anterior tibial artery
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Anomalous Muscles – 15%
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Intrauterine factors
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Left=Right, develops during third trimester, therefore external compression not likely to be a factor (despite popularity of theory)
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? interruption of normal development
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Associated with early amniocentesis (<week 11) = 10X (150X higher if leak occurs)
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anterior horn cell death (due to maternal enterovirus infection)
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Physical Examination
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Associated with UE, back, leg anomalies, ABN reflexes.
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reference with knee 90deg flexed.
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torsional alignment, varus, valgus, overall size and shape of leg, ankle, foot.
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usually shorter and wider than normal foot.
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check equinus in flexion and extension (account for gastroc & soleus)
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check resting varus / valgus as well as maximum correction
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check lateral border of foot (calcaneocuboid)
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n.b. check with foot dorsiflexed as well (avoid overcorrection = iatrogenic rocker bottom foot)
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check dorsolateral foot (talar head — should line up with patella)
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line up forefoot onto talar head
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all deformites should be assessed in relation to next proximal segment.
Radiographs
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tough to read, tough to reproduce – try to get in maximal correction with (simulated) weight bearing.
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On AP:
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AP talocalcaneal angle (usually < 20 deg in clubfoot)
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tarsal – first metatarsal angle (up to 30 degrees of valgus in norma foot, mild to severe varus in clubfoot)
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medial displacement of cuboid on calcaneus
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on lateral ( taken in max dorsiflexion & lat. rotation without pronation):
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talocalcaneal angle (usu. < 25 deg in clubfoot)
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talar – 1st MT angle (if plantar flexed, plantar soft tissues are contracted or midtarsal bony deformity is present
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MORE TO COME (ON TREATMENT AND RECURRENCE)